Author name: Anchal Negi

Vascular Surgery

DVT (Deep Vein Thrombosis) — Warning Signs, Risk Factors and Treatment

DVT — deep vein thrombosis — is a blood clot in the deep veins of the leg, and its symptoms are dangerously easy to ignore or mistake for a muscle problem. What makes DVT particularly serious is not the clot in the leg itself — it is what happens when a fragment breaks off and travels to the lungs. That is a pulmonary embolism, and it can be fatal within minutes. Dr. Ved Prakash, Director of CTVS and Vascular Surgery at Yatharth Super Speciality Hospitals, Greater Noida, explains DVT deep vein thrombosis symptoms, who is most at risk, and what treatment involves. What Is DVT — Deep Vein Thrombosis? Deep veins are the major vessels that carry blood from the legs back to the heart. When a clot forms in one of these deep veins — most commonly in the calf, thigh, or pelvis — blood cannot drain normally from the affected leg. The result is the swelling, pain, and warmth that characterise DVT deep vein thrombosis symptoms. And because the deep veins are connected directly to the pulmonary circulation, a piece of the clot can detach and reach the lungs at any time. DVT Deep Vein Thrombosis Symptoms — What to Look For Unilateral (One-Sided) Leg Swelling DVT almost always affects one leg. Swelling of both legs together is rarely caused by DVT — it more commonly indicates heart failure or low albumin. If one calf or thigh is noticeably larger than the other and this developed over hours or a few days, DVT must be ruled out urgently. Calf Pain and Tenderness Aching or tenderness in the calf — often mistaken for a muscle strain or cramp. The pain is typically worse when walking and when the foot is flexed upward (Homan’s sign — though this is not reliable as a standalone test). Many patients describe it as a persistent heaviness or soreness that does not improve with rest the way a muscle injury would. Warmth and Redness The skin over the clotted vein may feel warm to the touch and appear reddish or purplish — caused by the inflammatory response to the clot. This can look similar to cellulitis, which is a skin infection. DVT and cellulitis require completely different treatments, so a duplex ultrasound — not antibiotics alone — is always needed when the diagnosis is uncertain. Distended Surface Veins When the deep vein is blocked, blood reroutes through the surface veins — which then become visibly distended. This is different from pre-existing varicose veins and appears more suddenly. If you have sudden breathlessness, sharp chest pain, or are coughing blood — go to a hospital emergency immediately. These are pulmonary embolism symptoms and require urgent treatment. Who Is at Risk of DVT? Recent surgery — particularly orthopaedic surgery (hip or knee replacement), abdominal or pelvic surgery. Post-surgical DVT prevention with anticoagulants is standard practice in all major hospitals. Prolonged immobility — long-haul flights over 4 hours, extended bed rest, or a limb in plaster cast. Economy class travel over 8 hours increases DVT risk 2–3 fold. Cancer — many cancers activate the clotting system. DVT is sometimes the first sign of an undetected malignancy. Pregnancy and postpartum period — DVT risk is 5–10 times higher in pregnant women, particularly in the third trimester and the 6 weeks after delivery. Oral contraceptive pills or hormone replacement therapy — oestrogen increases clotting factor levels, particularly when combined with smoking. Inherited clotting disorders — Factor V Leiden, Protein C or S deficiency, antiphospholipid syndrome. Recurrent DVT without an obvious trigger should prompt testing for these. Post-COVID — COVID-19 infection causes a significant pro-thrombotic state. DVT incidence is elevated in patients recovering from COVID-19, particularly those who were hospitalised. Obesity and varicose veins — both impair venous return from the legs and increase background DVT risk. How Is DVT Diagnosed? Duplex Ultrasound The primary test. Non-invasive, painless, and highly accurate. Shows the clot location, how far it extends, and whether it is freely floating (mobile clots carry a higher embolism risk). This is the test that confirms or rules out DVT — not clinical examination alone. D-Dimer Blood Test A negative D-dimer in a low-probability patient effectively rules out DVT without needing an ultrasound. A positive D-dimer is not diagnostic on its own — it only means something is causing clotting activity (which could be DVT, infection, pregnancy, or many other conditions). Duplex ultrasound confirms. Wells Score A clinical scoring system that estimates DVT probability before imaging, based on symptoms, risk factors, and whether an alternative diagnosis is more likely. A high Wells score plus a positive D-dimer means DVT until proven otherwise by ultrasound. DVT Treatment Anticoagulation: The cornerstone of treatment. Blood thinners prevent the clot from growing and allow the body to dissolve it gradually. Modern DOACs — rivaroxaban or apixaban — are used in most cases and do not require regular INR blood monitoring, unlike warfarin. Duration is 3 months for provoked DVT (surgery, flight), 6 months or longer for cancer-related DVT or unprovoked recurrent episodes. Compression stockings: Reduce leg swelling and significantly lower the risk of post-thrombotic syndrome — the long-term venous damage that can follow an unresolved DVT clot. Catheter-directed thrombolysis: Reserved for large, limb-threatening DVTs causing severe swelling and circulatory compromise — a catheter delivers clot-dissolving drugs directly into the thrombosed vessel. IVC filter: A filter placed in the inferior vena cava to catch clots before they reach the lungs — used when anticoagulation is contraindicated (recent surgery, active bleeding). For patients with varicose veins and DVT — both conditions affecting the venous system — Dr. Ved Prakash treats both simultaneously at Yatharth Hospital to address the root cause of recurrent venous disease. Frequently Asked Questions — DVT Deep Vein Thrombosis Symptoms What are the classic DVT deep vein thrombosis symptoms? Unilateral leg swelling, calf pain and tenderness (worse on foot flexion), warmth and redness over the affected calf, and distended surface veins. More than 50% of DVTs are symptomless — which

Cost & Insurance

CGHS Coverage for Heart Surgery — What Central Government Employees Must Know

CGHS Coverage for Heart Surgery — CGHS, the Central Government Health Scheme, covers heart surgery for eligible central government employees, pensioners, and their dependants, but the coverage process has specific requirements that many patients only discover after treatment has begun. Understanding how CGHS works for cardiac procedures before your surgery date prevents delays, unexpected bills, and disputes with the CGHS office. Dr. Ved Prakash, Director of CTVS at Yatharth Super Speciality Hospitals, Greater Noida, explains what CGHS covers, how pre-authorisation works, and what patients need to know before bypass or valve surgery.   Who Is Eligible for CGHS Coverage for Heart Surgery? Central government employees in service (Group A, B, C, D) Central government pensioners and their spouses Dependent family members — defined as spouse, unmarried daughters, sons below 25 years, and parents residing with the beneficiary Members of Parliament, judges of Supreme Court and High Courts, and certain other constitutional bodies Freedom fighters covered under certain CGHS categories CGHS beneficiaries must hold a valid CGHS card to access treatment at empanelled hospitals. What Heart Procedures CGHS Covers Procedure CGHS Coverage Important Note Coronary Angiography Yes — covered at CGHS rates Referral letter from CGHS MO or specialist required Bypass Surgery (CABG) Yes — covered at CGHS package rates Pre-authorisation required for elective surgery Heart Valve Replacement (Mechanical) Yes — surgery + valve implant covered Valve brand may be specified — clarify before surgery Heart Valve Replacement (Biological) Partially — CGHS rate may not cover full biological valve cost Difference may be payable by patient TAVI Partially — CGHS rate is significantly below actual valve device cost Patient typically pays the valve cost difference Pacemaker Implantation Yes — at CGHS device rates Specific device models covered — ask hospital Paediatric Heart Surgery (ASD, VSD, TOF) Yes — covered for eligible dependent children Pre-authorisation and specialist referral required Varicose Vein EVLT Laser Yes — when symptomatic and medically indicated Cosmetic treatment not covered How CGHS Pre-Authorisation Works for Heart Surgery For any planned (elective) cardiac procedure, CGHS pre-authorisation must be obtained before admission. Without it, reimbursement claims can be delayed or partially rejected. Step 1 — Referral from CGHS Medical Officer A CGHS beneficiary first sees a CGHS Medical Officer (MO) at a CGHS Wellness Centre who issues a referral to a specialist — cardiologist or cardiac surgeon — at an empanelled hospital. Alternatively, in cities where cardiology specialists are available at CGHS polyclinics, referral may come from there. Step 2 — Specialist Recommendation Letter The cardiac surgeon at the empanelled hospital — in this case Dr. Ved Prakash — provides a formal recommendation letter stating the diagnosis, the procedure recommended, its urgency, and the estimated cost at CGHS package rates. Step 3 — Pre-Authorisation Application The patient or the hospital’s CGHS facilitation desk submits the pre-authorisation request to the Additional Director, CGHS, of the relevant city zone. Required documents: CGHS card copy, referral letter, specialist recommendation, angiogram or echocardiogram report, and the hospital’s CGHS empanelment proof. Step 4 — Approval and Admission Once pre-authorisation is received — typically within 3–7 working days for non-emergency cases — the patient is admitted for surgery. The hospital bills CGHS directly for covered procedures at the approved package rates. Emergency Surgery For emergency cardiac surgery — Type A aortic dissection, acute heart attack requiring urgent bypass, critical valve failure — treatment proceeds first. CGHS reimbursement is claimed retrospectively within 3 months of discharge. Emergency cases should have the CGHS card presented at admission regardless. What CGHS Package Rates Mean in Practice CGHS pays hospitals at fixed package rates that are periodically revised. These rates are often lower than a hospital’s standard market rates. What this means for patients: For bypass surgery — most empanelled hospitals accept CGHS rates and charge no balance billing for the standard procedure in a shared or semi-private room. For biological valve replacement — the CGHS rate for the valve implant may cover a basic tissue valve but not a premium bioprosthetic valve. The difference between the CGHS rate and the premium valve cost is sometimes payable by the patient. For TAVI — the transcatheter valve device costs significantly more than the CGHS package rate. Most CGHS patients who undergo TAVI pay a device cost difference of ₹3–8 lakhs depending on valve type. For room upgrades — CGHS covers treatment at the standard room category. Upgrading to a private room incurs additional daily room charges payable by the patient. ECHS — For Defence Personnel The Ex-Servicemen Contributory Health Scheme (ECHS) works similarly to CGHS for defence personnel. ECHS empanelment is separate from CGHS empanelment — a hospital empanelled under CGHS may or may not be empanelled under ECHS. Confirm ECHS empanelment at +91-9355255106 before booking. If you have a CGHS card and have been recommended bypass surgery or valve surgery, or if you want an independent view of your angiogram before proceeding, a cardiac second opinion can be obtained by sharing your reports via WhatsApp — at no cost for the initial pre-assessment. Frequently Asked Questions — CGHS Heart Surgery Coverage Does CGHS cover bypass surgery? Yes — CABG is covered at CGHS package rates at empanelled hospitals. Pre-authorisation is required for elective surgery. Emergency bypass is treated first and reimbursed retrospectively. Does CGHS cover TAVI surgery? Partially. CGHS covers TAVI as a procedure but at a package rate significantly below the actual transcatheter valve device cost. Most patients pay the valve cost difference — clarify this with the hospital billing team before scheduling. How do I get CGHS pre-authorisation for heart surgery? Get a referral from your CGHS MO, obtain a recommendation letter from the cardiac surgeon at the empanelled hospital, and submit a pre-authorisation request to the Additional Director, CGHS, of your city zone. The hospital’s CGHS desk assists with this process. Is Yatharth Hospital Greater Noida empanelled under CGHS? Yatharth Super Speciality Hospitals, Greater Noida accepts CGHS and ECHS patients for cardiac surgery. Confirm current empanelment status for your specific procedure by calling +91-9355255106 before booking.

Paediatric Heart Surgery

Tetralogy of Fallot — What Parents Need to Know After a TOF Diagnosis

What Parents Need to Know After a TOF Diagnosis: Tetralogy of Fallot — TOF — is the most common cyanotic congenital heart defect, and a diagnosis that most parents receive with no preparation and very little information about what comes next. If your baby has just been diagnosed with TOF, this guide explains what it means, what symptoms to watch for at home, what surgery involves, and — critically — what your child’s life looks like after repair. Dr. Ved Prakash, Director of CTVS at Yatharth Super Speciality Hospitals, Greater Noida, has performed TOF repairs from early infancy across Medanta, Narayana, and Yatharth Hospital. What Is Tetralogy of Fallot? Tetralogy of Fallot is four structural abnormalities that occur together in the same heart. Understanding all four is important because they interact — each one making the others worse: A large VSD (ventricular septal defect) — a large hole between the two lower chambers, allowing blue (deoxygenated) blood to mix with red (oxygenated) blood. Pulmonary stenosis — narrowing of the outflow from the right ventricle to the lungs. This obstruction means less blood reaches the lungs to pick up oxygen, and more blue blood is pushed through the VSD to the body instead. Overriding aorta — the aorta sits directly over the VSD rather than exclusively over the left ventricle, receiving a mix of blue and red blood from both chambers. Right ventricular hypertrophy — the right ventricle thickens because it is pumping against the obstructed pulmonary outflow. This worsens over time if repair is delayed. The net result: the body continuously receives blood that has not been fully oxygenated — causing the characteristic blue discolouration (cyanosis) of Tetralogy of Fallot. Tetralogy of Fallot Symptoms — What Parents See Cyanosis — The Blue Baby Appearance Blue or purple discolouration of the lips, tongue, fingernails, and toenails. In some babies with TOF, cyanosis is obvious at birth. In others — particularly where the pulmonary stenosis is mild — cyanosis develops gradually in the first weeks of life as the ductus arteriosus (a foetal blood vessel that keeps oxygen supply adequate in the womb) closes after birth. A baby who was pink at birth and becomes increasingly blue in the first month needs urgent cardiac assessment. Tet Spells — The Most Frightening Symptom A tet spell is a hypercyanotic episode — a sudden, dramatic worsening of cyanosis triggered by anything that decreases pulmonary blood flow: crying, feeding, defecation, waking from sleep, or even nothing obvious. During a tet spell the baby turns intensely blue, becomes extremely distressed and inconsolable, breathes very fast, and may go limp or briefly lose consciousness from cerebral hypoxia. Tet spells are caused by spasm of the right ventricular outflow tract — the already-narrowed channel from the right ventricle to the lungs temporarily clamps shut, redirecting almost all right-sided output through the VSD to the body. Almost no blood reaches the lungs. Oxygen saturation plummets. If your baby has a tet spell: pull the knees firmly up to the chest (squatting position for an infant), keep the baby calm, and call emergency services immediately. Tet spells require hospital assessment and accelerate the need for surgery. Do not wait to see if it settles. Squatting in Older Unrepaired Children Older children with unrepaired or partially palliated TOF instinctively squat after physical activity. Squatting compresses the femoral arteries — increasing systemic vascular resistance and reducing the right-to-left shunt through the VSD — temporarily pushing more blood to the lungs and relieving cyanosis. It is one of the most specific clinical signs in all of paediatric cardiology. Other Signs Poor growth and weight gain — the body cannot support normal growth when oxygen delivery is chronically reduced Exercise intolerance in older unrepaired children — breathlessness and cyanosis with minimal exertion Clubbing of fingers and toes — develops after months of chronic cyanosis How Tetralogy of Fallot Is Diagnosed Foetal echocardiogram: TOF is detectable at the 18–20 week anomaly scan in many cases — allowing planned delivery at a centre with paediatric cardiac surgery capability. Neonatal echocardiogram: Confirms the anatomy — size of the VSD, degree of pulmonary stenosis, position of the aorta, and right ventricular size. Pulse oximetry screening: Low oxygen saturation detected on routine newborn screening triggers further investigation. CT cardiac angiography: Used before surgery when the pulmonary artery anatomy needs detailed 3D mapping — particularly important for the branch pulmonary arteries which can be hypoplastic in severe TOF. Tetralogy of Fallot Surgery — What Is Done Complete surgical repair of TOF is performed under general anaesthesia and cardiopulmonary bypass. The operation addresses all the abnormalities simultaneously: VSD closure: The hole between the ventricles is closed with a patch — stopping the mixing of blue and red blood completely. Relief of pulmonary stenosis: The narrowed right ventricular outflow tract is widened by resecting obstructing muscle bundles, and often by placing a patch across the outflow tract or pulmonary valve to enlarge it. If the pulmonary valve itself is severely abnormal it may be removed — leaving the outflow tract open (transannular patch). This effectively relieves obstruction but results in pulmonary regurgitation. Result: Blue blood now goes through the pulmonary artery to the lungs normally. Oxygenated blood returns and goes through the aorta to the body. Cyanosis resolves. Surgery is typically performed between 3 and 6 months of age. If tet spells are occurring before this age, surgery is brought forward without delay. Results at experienced centres: operative mortality below 2–3%, complete repair achieved in virtually all cases. Life After TOF Surgery — The Honest Picture This is what parents most need to understand — and what most articles do not explain. The good news: The vast majority of children repaired in infancy grow up completely normally. They attend mainstream school, participate in sport, develop normally in every way. Most go on to live independent adult lives — working, driving, having children of their own. What requires lifelong monitoring: Annual echocardiogram — monitoring right ventricular size and function over time Pulmonary

Aortic Surgery

What Is an Aortic Aneurysm? Silent, Dangerous — and Treatable

An aortic aneurysm is a ballooning in the wall of the aorta — the body’s main artery — and the most dangerous thing about it is that it causes no symptoms until it is on the verge of rupturing. Rupture of an aortic aneurysm is one of the most lethal events in medicine, with mortality exceeding 80% even with emergency surgery. The goal — always — is to find it and treat it before that moment. Dr. Ved Prakash, Director of CTVS at Yatharth Super Speciality Hospitals, Greater Noida, explains what causes an aortic aneurysm, when it needs surgery, and what the surgical options are. What Is an Aortic Aneurysm? The aorta is the largest artery in the body — rising from the heart, arching through the chest, and running down through the abdomen to supply the entire lower body. The normal aortic diameter is approximately 2.0–3.0 cm. An aortic aneurysm is an abnormal widening of the aorta to more than 1.5 times its normal diameter — defined as greater than 3.0 cm in the abdominal aorta or greater than 4.5 cm in the thoracic aorta. As an aortic aneurysm enlarges, the aortic wall becomes progressively thinner and weaker. At a critical size, the wall can rupture — and the outcome is catastrophic. Types of Aortic Aneurysm Abdominal Aortic Aneurysm (AAA) The most common type — a ballooning in the portion of the aorta that runs through the abdomen. AAAs occur primarily in men over 65 with a history of smoking, high blood pressure, and high cholesterol. They are almost always discovered incidentally on an ultrasound or CT scan ordered for something else entirely — because they rarely cause symptoms. Thoracic Aortic Aneurysm (TAA) Aortic aneurysm in the chest — affecting the ascending aorta (rising from the heart), the arch, or the descending aorta. TAAs are more commonly associated with genetic conditions including Marfan syndrome and bicuspid aortic valve, and can affect patients at younger ages than AAAs. What Causes an Aortic Aneurysm? Atherosclerosis — the primary cause of AAA. Years of high blood pressure, high cholesterol, and smoking progressively weaken the aortic wall. Hypertension — the most important modifiable risk factor. Controlling blood pressure directly slows aneurysm growth. Smoking — doubles aortic aneurysm risk and accelerates growth rate significantly. The single most effective prevention is smoking cessation. Marfan syndrome and related connective tissue disorders — inherently weak aortic wall tissue; aneurysms can develop at younger ages and at smaller sizes than in the general population. Bicuspid aortic valve — associated with progressive ascending aortic enlargement; regular echocardiographic surveillance of the aortic diameter is essential. Family history — first-degree relatives of AAA patients have a 10–15% lifetime risk; screening ultrasound is recommended. Aortic Aneurysm Symptoms — Why It Is Called a Silent Killer The vast majority of aortic aneurysms produce no symptoms until they are large or rupturing. When symptoms do occur: Deep, constant back or abdominal pain — often mistaken for musculoskeletal pain. In the context of a known aortic aneurysm, this is a warning sign of rapid expansion or contained leak — requiring emergency assessment. Pulsating abdominal mass — a pulsating sensation in the centre of the abdomen, felt by the patient or detected on physical examination. Most clearly felt in thin patients. Hoarseness or difficulty swallowing — from a thoracic aneurysm pressing on the recurrent laryngeal nerve or oesophagus. Sudden, severe tearing abdominal or back pain with collapse is rupture until proven otherwise — call emergency services immediately. This is different from the presentation of aortic dissection, which classically causes chest pain — though both are surgical emergencies. When Does an Aortic Aneurysm Need Surgery? The decision is based primarily on size — larger aneurysms have exponentially higher annual rupture risk: AAA Diameter Annual Rupture Risk Recommendation 3.0–4.4 cm <0.5% Ultrasound surveillance every 12 months 4.5–5.4 cm 1–5% Surveillance every 6 months + surgical review 5.5 cm and above 10–25% Surgery recommended Growing >1 cm per year (any size) High Surgery recommended regardless of size EVAR vs Open Surgery — What Are the Options? EVAR — Endovascular Aneurysm Repair A stent-graft is delivered through the femoral arteries in the groin and positioned inside the aneurysm, lining it and excluding it from the circulation. No abdominal incision. Hospital stay 2–3 days. Recovery 2–3 weeks. Requires annual CT surveillance to check the stent-graft remains in position and there is no re-pressurisation (endoleak). Suitable for most infrarenal AAAs with appropriate anatomy. Open Surgical Repair The aneurysm is exposed through an abdominal incision, clamped, and replaced with a synthetic Dacron graft. More invasive — hospital stay 7–10 days, recovery 6 weeks — but provides a definitive, lifelong repair that does not require ongoing CT surveillance. Required when anatomy is not suitable for EVAR, or for juxtarenal/suprarenal aneurysms. TEVAR — Thoracic Endovascular Aortic Repair The endovascular equivalent of EVAR for descending thoracic aortic aneurysms — a stent-graft relined the diseased thoracic aorta through femoral artery access. For patients in Delhi NCR with a newly diagnosed or enlarging aortic aneurysm, aortic surgery in Delhi NCR at Yatharth Hospital covers the full range — EVAR, TEVAR, and open surgical repair. CT reports can be shared via WhatsApp for a pre-assessment before your visit. Frequently Asked Questions — What Is Aortic Aneurysm What is an aortic aneurysm and is it always fatal? An aortic aneurysm is an abnormal ballooning of the aortic wall. It is not immediately fatal — most detected aneurysms are small and slow-growing and can be treated safely with elective surgery before rupture. A ruptured aneurysm carries mortality above 80%. Finding it early is what saves lives. At what size does an aortic aneurysm need surgery? Surgery is generally recommended when an abdominal aortic aneurysm reaches 5.5 cm or when it grows more than 1 cm in 12 months. Below this, surveillance ultrasound every 6–12 months is the standard approach. What is EVAR and how is it different from open surgery? EVAR delivers a stent-graft through the groin without opening the

Uncategorized

What Is TAVI? Aortic Valve Replacement Without Opening the Chest

Aortic Valve Replacement Without Opening the Chest: TAVI — Transcatheter Aortic Valve Implantation — is a procedure that replaces a diseased aortic heart valve through a catheter passed through the leg artery, without any chest incision and without stopping the heart. For the millions of elderly patients in India who have been told their aortic valve is severely narrowed but that open surgery is too risky at their age, TAVI is often the answer. Dr. Ved Prakash, Director of CTVS at Yatharth Super Speciality Hospitals, Greater Noida, explains what TAVI is, how the procedure works, and crucially, how the decision between TAVI and open surgery is made. Why Was TAVI Developed? Aortic stenosis — narrowing of the aortic valve — is the most common serious valve disease in patients over 65. Once symptoms appear, the average untreated survival is 1–3 years. For decades, the only treatment was open surgical valve replacement, which carries significant risk in frail, elderly, or medically complex patients. The TAVI procedure was developed specifically to treat this group — offering a life-saving valve replacement without the trauma of open surgery. First performed in humans in 2002, TAVI has now been performed on over a million patients worldwide with results that equal or exceed open surgery in high-risk groups. What Is Aortic Stenosis — the Condition TAVI Treats? The aortic valve sits between the left ventricle and the aorta, controlling blood flow from the heart to the rest of the body. In aortic stenosis, the valve leaflets thicken, stiffen, and calcify over years — narrowing the valve opening until blood can no longer flow freely. The heart works harder to push blood through the narrowed valve, eventually leading to heart failure. Symptoms include breathlessness on exertion, chest pain, and blackouts (syncope). Once any of these symptoms appear, urgent treatment is needed. What Is TAVI — How the Procedure Works The TAVI procedure delivers a replacement biological tissue valve to the heart entirely through blood vessels — most commonly the femoral artery in the groin. Here is what happens: Transfemoral Access A small puncture is made in the femoral artery in the groin. No chest incision. A guidewire is advanced through the artery, up the aorta, and across the diseased aortic valve under continuous X-ray guidance. Valve Delivery The replacement valve — compressed onto a small expandable frame — is loaded onto a catheter and advanced over the guidewire to the level of the native aortic valve. Precise positioning is confirmed using simultaneous echocardiography and fluoroscopy. Valve Deployment The new valve is expanded — either by balloon inflation or by self-expansion depending on the valve type. It immediately opens and begins functioning, pushing the old calcified leaflets aside. The left ventricle now pumps blood freely through the new valve. Confirmation and Closure Echocardiography and angiography confirm the valve is working correctly and there is no significant leakage. The groin access site is closed with a vascular closure device — no surgical cut-down needed in most cases. Total procedure time: 1–2 hours. How Is the Decision Made — TAVI or Open Surgery? This is the question every patient and family deserves a clear answer to. At Yatharth Hospital, this decision is never made by one doctor alone — it is made by a Heart Team consisting of Dr. Ved Prakash (cardiac surgeon), an interventional cardiologist, and a cardiac anaesthetist, who review the echocardiogram, CT angiography, and clinical status together. Factor Favours TAVI Favours Open Surgery (SAVR) Age Over 75 Under 65–70 Surgical risk score High or intermediate Low Lung function Poor (COPD) Normal Aortic calcification Severe (avoids clamping) Minimal Need for bypass surgery simultaneously No Yes — open surgery required Valve durability need Shorter life expectancy Young patient needing 20+ years What to Expect After TAVI Hospital stay: 3–5 days in most patients — compared to 7–10 days after open surgery Walking: The following day after TAVI in most cases Breathlessness: Most patients notice dramatic improvement within days as the heart begins pumping freely through the new valve Pacemaker risk: Approximately 10–15% of patients require a permanent pacemaker — this is discussed before every procedure Medications: Aspirin lifelong. Clopidogrel for 3–6 months. No warfarin required unless the patient has atrial fibrillation. Follow-up: Echocardiogram at 1 month, 1 year, then annually to monitor valve function TAVI valve lifespan: 10–15 years in most patients. A second TAVI (valve-in-valve) can often be performed if the first valve eventually wears out. For patients in Delhi NCR and surrounding areas, TAVI at Yatharth Hospital is performed by Dr. Ved Prakash with a dedicated Heart Team and full cath lab and hybrid OT capability. For more on heart valve surgery options including both TAVI and open procedures, visit the heart valve surgery page. Key Aspects of TAVI / TAVR (Explained Simply) 1. Procedure TAVI (or TAVR) is a minimally invasive heart procedure.Instead of opening the chest like traditional open-heart surgery, doctors make a small puncture (about 1 cm) usually in the groin artery. A thin tube called a catheter is gently guided through the blood vessels to reach the heart. 2. Valve Replacement A new artificial valve, folded inside the catheter, is delivered to the damaged aortic valve.Once in position, the new valve expands and pushes the old valve aside, immediately allowing blood to flow normally again.  3. Who Needs TAVI? TAVI mainly treats severe aortic stenosis, a condition where the heart valve becomes narrow or stiff and blocks blood flow.It can be suitable for patients who are high-risk, intermediate-risk, or even low-risk for traditional open-heart surgery, depending on medical evaluation. 4. Benefits of TAVI No large chest incision Shorter hospital stay (usually 2–3 days) Less pain and faster recovery Earlier return to normal daily activities  5. Possible Risks Like any medical procedure, TAVI has some risks, although it is safe for most patients: Bleeding or infection Stroke (rare) Heart rhythm problems Small leak around the new valve Frequently Asked Questions — What Is TAVI Procedure What is TAVI and how is it different

Vascular Surgery

Peripheral Artery Disease Symptoms — Why Leg Pain While Walking Is a Warning Sign

Peripheral artery disease symptoms are one of the most under-recognised warning signs in Indian medicine — and in the diabetic population, which is enormous across UP, Delhi NCR, and the surrounding regions, peripheral artery disease often presents with no warning at all until the disease is critical. Dr. Ved Prakash, Director of CTVS and Vascular Surgery at Yatharth Super Speciality Hospitals, Greater Noida, explains what peripheral artery disease is, the stages of symptoms from mild to limb-threatening, and when intervention is urgently needed. What Is Peripheral Artery Disease? Peripheral artery disease (PAD) is atherosclerosis — plaque buildup — affecting the arteries that supply blood to the legs. It is the same disease process as coronary artery disease in the heart, occurring in a different location. As the arteries narrow, the leg muscles do not receive enough blood during exercise — and as the disease progresses, not even at rest. PAD affects approximately 200 million people worldwide and is significantly underdiagnosed in India. PAD is not the same as deep vein thrombosis or varicose veins. Those are venous (vein) problems. PAD is an arterial (artery) problem — it is about blood not reaching the leg, not blood failing to drain from it. The distinction matters because the symptoms, investigations, and treatments are completely different. For comparison, read about deep vein thrombosis. Peripheral Artery Disease Symptoms — Stage by Stage Stage 1 — Claudication The hallmark PAD symptom. Cramping, tightness, or aching in the calf, thigh, or buttock that comes on after a predictable walking distance — say, 300 metres — and stops you in your tracks. After 5–10 minutes of standing still, the pain disappears completely. You can then walk again — until the same distance is reached. The location of the pain tells you the level of arterial disease: calf claudication means disease in the superficial femoral artery (mid-thigh level); thigh and buttock claudication means disease in the iliac arteries of the pelvis. Claudication is often written off as arthritis, age-related muscle weakness, or sciatica — none of which share its specific pattern of predictable onset with walking and complete relief with rest. Stage 2 — Worsening Claudication As peripheral artery disease progresses without treatment, the claudication distance shortens. A patient who could walk 500 metres two years ago now manages 100. This reflects worsening arterial narrowing and is the signal that intervention should be considered before the next stage develops. Stage 3 — Rest Pain When peripheral artery disease becomes severe enough, the foot no longer receives adequate blood even lying in bed. The patient experiences burning or aching pain in the foot and toes — classically worse at night, and relieved slightly by hanging the foot over the edge of the bed (gravity assists blood flow). Rest pain is a medical emergency. Without treatment, tissue loss — gangrene — follows within weeks. Stage 4 — Tissue Loss (Ulceration or Gangrene) The most advanced peripheral artery disease symptom. Wounds on the foot or toes that do not heal despite weeks of dressing, or areas of blackened, dry or wet gangrene. In diabetic patients, this is often the first and only presentation of peripheral artery disease — because diabetic neuropathy has eliminated the earlier pain warning signals entirely. Peripheral Artery Disease in Diabetic Patients — The Silent Crisis This is the most important section for a significant proportion of readers across western UP and Delhi NCR, where diabetes rates are among the highest in the country. Diabetic neuropathy — nerve damage from chronic high blood sugar — destroys the pain sensation in the feet and lower legs. A diabetic patient with PAD advanced enough to cause rest pain may feel nothing. A small cut on the foot from a tight shoe may go unnoticed for days. A wound that should heal in a week sits open for months — because the blood supply needed for healing is not arriving. By the time a diabetic patient presents to a vascular surgeon with a non-healing foot wound, they often have severe, critical peripheral artery disease that has been silently progressing for years. Annual ABI (Ankle-Brachial Index) measurement is recommended for all diabetic patients over 50. It takes five minutes, is completely painless, and can detect PAD before any symptoms develop. How Is Peripheral Artery Disease Diagnosed? ABI (Ankle-Brachial Index): Blood pressure cuff at the ankle compared to the arm. A ratio below 0.9 confirms PAD. Below 0.5 indicates critical ischaemia. Fast, painless, available in any vascular clinic. Duplex Doppler ultrasound: Maps the arterial anatomy and identifies narrowings without radiation or dye. CT angiography: Detailed 3D imaging from the aorta to the foot — essential before planning angioplasty or bypass surgery. Conventional angiography: Catheter-based — performed in the interventional suite when treatment is planned immediately. Peripheral Artery Disease Treatment Medical management: Antiplatelet drugs (aspirin or clopidogrel), statin therapy, strict blood pressure and blood sugar control, and smoking cessation. Supervised walking exercise programmes — counterintuitive but effective — can increase the claudication distance by 50–100% in 3 months. Endovascular treatment (angioplasty/stenting): A catheter opens the narrowed artery and a stent holds it open. Minimally invasive, 1–2 day hospital stay, faster recovery. Peripheral bypass surgery: For long or multiple blockages not amenable to angioplasty — a vein or synthetic graft bypasses the blocked arterial segment. More invasive but durable, with recovery of 4–6 weeks. For patients with critical limb ischaemia or a non-healing diabetic foot wound, peripheral vascular disease treatment in Delhi NCR at Yatharth Hospital is available on an urgent basis — WhatsApp your clinical photos and report to +91-9355255106 for a rapid pre-assessment. Frequently Asked Questions — Peripheral Artery Disease Symptoms What is the most common symptom of peripheral artery disease? Claudication — leg cramping that starts after a predictable walking distance and disappears completely with 5–10 minutes of rest. It affects about 50% of PAD patients; the other half are asymptomatic, particularly diabetics. Why are diabetic patients at higher risk? Diabetes doubles PAD risk and eliminates the pain signals (via neuropathy)

Vascular Surgery

EVLT Laser Treatment for Varicose Veins — What to Expect Before, During and After

EVLT — Endovenous Laser Treatment — is a walk-in, walk-out procedure that closes varicose veins permanently from the inside using laser energy, with no surgical incision, no stitches, and no general anaesthesia. It has replaced surgical stripping as the first-choice treatment for most varicose veins. Dr. Ved Prakash, Director of CTVS and Vascular Surgery at Yatharth Super Speciality Hospitals, Greater Noida, explains exactly what EVLT laser treatment involves, who it works for, and what recovery looks like day by day. How Does EVLT Work? To understand EVLT, you first need to understand the problem it solves. Varicose veins are caused by valve failure in the long saphenous vein — the main superficial vein that runs from the foot to the groin. When this vein’s valves stop working, blood pools and the branches feeding off it become the visible varicose veins on the surface. EVLT targets the root cause directly. A laser fibre is inserted into the long saphenous vein through a needle puncture — no cut, no incision. As the fibre is slowly withdrawn along the length of the vein, it delivers controlled laser energy to the vein wall, causing it to collapse and seal permanently. Within 4–8 weeks, the sealed vein is absorbed by the body. Blood that previously pooled in the diseased vein automatically reroutes through the healthy deep veins — relieving symptoms and collapsing the visible surface varicosities. What Happens on the Day of EVLT Duplex Ultrasound Mapping First Before the laser is inserted, a duplex ultrasound scan is performed to map the incompetent saphenous vein — its length, diameter, and the location of the incompetent junction at the groin. This guides precisely where the laser fibre will be placed and how long it needs to run. Access Under Ultrasound Guidance A fine needle puncture — usually at the knee — is made into the saphenous vein under ultrasound vision. No incision, no stitch. A thin sheath is introduced through which the laser fibre is threaded up to the level of the groin junction. Tumescent Anaesthesia A dilute local anaesthetic solution is injected around the vein using very fine needles along its entire length. This is called tumescent anaesthesia. It numbs the vein completely — so the laser is painless — and also acts as a heat buffer protecting the surrounding tissue from thermal injury. This step takes 10–15 minutes and is the only mildly uncomfortable part of the procedure. Laser Activation The laser is switched on and the fibre is slowly pulled back along the vein at a controlled rate. You feel nothing. The procedure takes 15–25 minutes per leg. Compression and Walking A compression bandage is applied immediately. Patients are asked to walk for 20–30 minutes in the corridor before leaving the hospital. Walking straight away activates the deep veins and prevents clot formation. You then go home — same day, every time. Recovery After EVLT — Day by Day Day of procedure: Walk 20–30 minutes before leaving. Go home. Mild tightness along the treated vein is normal — this is the vein sealing. Not painful. Days 1–3: Bruising develops along the vein track. This is expected and not a complication. Continue walking 30 minutes daily. Compression stocking on throughout the day, off at night. Days 3–5: Most desk-work patients return to work. The vein feels like a cord under the skin — the sealed vein being absorbed. This resolves over 4–6 weeks. Week 1: Avoid hot baths, saunas, swimming. Shower normally. No prolonged standing for more than 2 hours at a stretch. Week 2: Bruising fades significantly. Compression stocking continues for the full 2 weeks post-procedure. Week 4–6: Follow-up duplex ultrasound confirms the vein is fully closed. Any residual smaller tributary veins visible at the surface are treated with sclerotherapy injection at this visit if needed. EVLT vs Surgical Stripping — Why EVLT Is Now the Standard Feature EVLT Laser Surgical Stripping Anaesthesia Local (tumescent) General or spinal Incisions None — needle puncture only 2–3 small incisions Same-day discharge Yes — every time Usually day procedure or overnight Return to desk work 1–2 days 2–3 weeks Post-procedure pain Mild tightness only Wound pain for 5–10 days 5-year closure rate >90% >85% — similar Who Qualifies for EVLT? EVLT is suitable for most patients with symptomatic varicose veins from great or small saphenous vein incompetence. A duplex ultrasound assessment beforehand confirms suitability — specifically, the vein must be large enough to accept the laser fibre and accessible by needle puncture. Very large or tortuous veins may require surgical stripping. Active DVT in the affected leg is a contraindication. To understand more about the underlying condition EVLT treats, read about how varicose veins develop. For more on all treatment options — including sclerotherapy and surgical alternatives — visit our varicose vein treatment page. Frequently Asked Questions — EVLT Laser Varicose Vein Treatment Is EVLT laser treatment for varicose veins painful? No — the tumescent anaesthesia numbs the vein completely before the laser activates. Patients feel no pain during the procedure. Mild tightness and bruising along the treated vein is normal for 3–7 days after and is not painful. How long does recovery take after EVLT? Desk-work patients return within 1–2 days. Walking from the same day. Compression stockings for 2 weeks. Physically demanding work — prolonged standing or heavy lifting — avoided for 2 weeks. Full activity by week 3. How long does EVLT last? The treated vein is permanently closed. Closure rates exceed 90% at 5 years. The treated vein does not reopen. New varicose veins can develop from other branches over subsequent years but the treated vein stays sealed. Can both legs be treated with EVLT on the same day? Yes — bilateral EVLT on the same day is safe and commonly performed. Post-procedure walking is required. Most patients tolerate bilateral EVLT well under tumescent anaesthesia. Dr. Ved Prakash | Director, CTVS — Yatharth Super Speciality Hospitals, Greater Noida 📞 +91-9355255106  | 📧 drvedprakash@gmail.com  | Book Appointment →

Vascular Surgery

Varicose Veins — Causes, Symptoms and When They Become Dangerous

Varicose veins causes symptoms: Varicose veins are not just a cosmetic concern — they are a progressive condition caused by damaged vein valves, and without treatment they tend to worsen over time. Many patients visit a vascular specialist only after years of ignoring early warning signs, assuming them to be a normal part of ageing. Dr. Ved Prakash, Director of CTVS and Vascular Surgery at Yatharth Super Speciality Hospitals, explains what causes varicose veins, how symptoms appear at different stages, and when the condition becomes medically serious. What Are Varicose Veins? Your leg veins carry blood from the feet back up to the heart — against gravity. To stop blood from flowing backwards between heartbeats, veins contain one-way valves that open as blood flows upward and snap shut as soon as the heartbeat stops pushing. When these valves weaken or are damaged, blood pools in the vein between beats. The pooled blood stretches the vein wall — and over time the vein enlarges, twists, and becomes visibly distorted under the skin. This is a varicose vein. It is always caused by valve failure — not by the pressure of standing, not by diet, and not by crossing your legs. Varicose Veins Causes — Why They Develop Prolonged Standing or Sitting The most significant occupational risk factor. Teachers, nurses, surgeons, security guards, factory floor workers, and retail staff who stand for 6 or more hours daily put sustained pressure on their leg vein valves. Over years this weakens the valves. Desk workers who sit for long periods face a different but related problem — pelvic vein compression impairs drainage from both legs. Family History If one parent has varicose veins, your lifetime risk is approximately 40%. If both parents do, it rises to 90%. Valve weakness is largely inherited — it is not something you caused. Pregnancy Multiple mechanisms combine during pregnancy: blood volume increases by 40–50%, the hormone progesterone relaxes vein walls, and the growing uterus compresses the pelvic veins — all increasing the pressure on leg vein valves. Varicose veins from a first pregnancy often improve partially after delivery. They typically worsen with subsequent pregnancies. Obesity Increased intra-abdominal pressure from excess body weight continuously impairs blood return from the legs — accelerating valve deterioration. Age Valve elasticity decreases naturally with age. Most symptomatic varicose veins present in patients over 40. Previous DVT A past episode of deep vein thrombosis can permanently damage vein valves — causing post-thrombotic varicose veins that are more difficult to treat and more likely to cause skin complications. Common Symptoms of Varicose Veins  Varicose veins are usually visible on the legs, but they can also make your legs feel uncomfortable. Common signs include: Visible VeinsYou may notice enlarged veins that look blue or dark purple. These veins often appear twisted, raised, or bulging under the skin, mainly on the legs. Leg Pain or HeavinessYour legs may feel tired, sore, or heavy, especially after standing or sitting for a long time. SwellingMild swelling can develop around the feet, ankles, or lower legs, particularly by the end of the day. Skin Irritation or ChangesThe skin around the veins may itch, burn, or feel warm. If not treated, the skin color may slowly become darker. Leg CrampsSome people experience muscle cramps or tightening in the legs, often during the night while sleeping. Varicose Veins Symptoms — Staged by Severity Varicose veins progress through a clinically recognised staging system called CEAP classification. Understanding where you are in this progression determines urgency of treatment. C1 — Spider Veins (Telangiectasia) Small, thread-like red or purple veins visible very close to the skin surface. No symptoms — purely cosmetic. Common in women from their 20s onward. C2 — Varicose Veins Bulging, rope-like veins visible under the skin — typically on the calf, back of the knee, or inner thigh. May or may not cause symptoms at this stage. Many patients live with C2 disease for years before seeking help. C3 — Swelling (Oedema) Ankle and lower leg swelling that builds through the day and does not fully resolve overnight. This indicates significant venous hypertension — the vein pressure has risen to the point where fluid is being pushed out into the surrounding tissue. At this stage, symptoms include leg heaviness, aching after standing, and cramping at night. C4 — Skin Changes Brown pigmentation (haemosiderin staining) around the ankle, eczema over the varicose veins, or a hardening and tightening of the skin above the ankle (lipodermatosclerosis). These are signs of longstanding venous hypertension damaging the skin. This stage requires treatment — it is no longer purely cosmetic. C5 — Healed Venous Ulcer A venous ulcer near the inner ankle that has previously healed but left a scar. These patients are at high risk of recurrence without treatment of the underlying varicose veins. C6 — Active Venous Ulcer An open wound near the inner ankle that does not heal despite weeks of dressing. This is a medical emergency in vascular terms — it will not close without treating the underlying varicose veins driving the venous hypertension. Referral to a vascular surgeon is urgent. Primary Causes and Risk Factors of Varicose Veins Varicose veins develop when the tiny valves inside your veins stop working properly. These valves normally help blood flow upward toward the heart. When they weaken or get damaged, blood flows backward and collects in the veins, making them swollen and visible.  Main Causes and Risk Factors Age (Getting Older)As we age, veins naturally lose strength and flexibility. The valves become weaker, increasing the chance of varicose veins. PregnancyDuring pregnancy, the body produces more blood to support the baby. Hormonal changes and pressure from the growing uterus put extra strain on leg veins, causing them to stretch. Gender (Women at Higher Risk)Women develop varicose veins more often than men. Hormonal changes during pregnancy, menopause, or while using birth control pills can affect vein health. Obesity or Excess WeightExtra body weight puts additional pressure on leg veins, making it harder for blood to

Cardiac Surgery

What Is Coronary Angiography? Procedure, Results and What Happens Next

Coronary angiography is the definitive test for identifying blockages in the heart arteries — the investigation that gives every cardiologist and cardiac surgeon the exact road map needed to plan the right treatment. If you have been referred for coronary angiography, or if you already have a report and cannot make sense of it, Dr. Ved Prakash, Director of CTVS at Yatharth Super Speciality Hospitals, Greater Noida, explains what the procedure involves, what the results mean, and what comes next.   What Is Coronary Angiography? Coronary angiography is a catheter-based procedure in which a special dye (contrast agent) is injected directly into the coronary arteries while X-ray images are captured continuously. The dye makes the inside of the arteries visible — showing exactly where blockages are, how severe they are, and how many arteries are affected. It is performed in a cardiac catheterisation laboratory (cath lab), takes 30–45 minutes, and is done under local anaesthesia. You remain awake throughout. Most hospitals now use the radial artery in the wrist for access — which means less discomfort and same-day or next-morning discharge in most cases. Why Has Your Doctor Recommended Coronary Angiography? Coronary angiography is recommended when: Chest pain on exertion suggests blocked coronary arteries (angina) An ECG, stress test, or echocardiogram has shown changes that need further investigation You are being assessed before major heart surgery — such as valve replacement — and need coronary status confirmed You have had a heart attack and require urgent identification of the blocked artery A CT coronary angiogram has found blockages that need catheter-based confirmation before treatment What Happens During Coronary Angiography — Step by Step Preparation You fast for 4–6 hours before the procedure. An IV line is placed in your arm. The access site — usually your wrist — is cleaned and numbed with local anaesthetic. Catheter Insertion A thin, flexible catheter is inserted into the radial artery and guided up through the arm and chest to the opening of each coronary artery. This is done under live X-ray guidance. You feel pressure but no pain. Dye Injection and Imaging A small amount of contrast dye is injected into each coronary artery. For 10–15 seconds you will feel a warm flush through the chest — this is completely normal and passes quickly. X-ray images are captured from multiple angles as the dye flows through the arteries. Catheter Removal and Recovery The catheter is removed. A compression band is placed over the wrist access site. Within an hour you can sit up and eat. Most patients go home the same evening or the following morning. How to Read Your Coronary Angiography Report This is where most patients feel lost — and where the most important decisions get made. Here is what the numbers mean. Percentage Blockage (Stenosis) Stenosis What It Means Typical Action 0–49% Mild narrowing — does not significantly restrict flow Medication and lifestyle. No procedure needed. 50–69% Moderate — may or may not restrict flow FFR pressure wire test to confirm significance 70–90% Significant — restricts blood flow Treatment recommended: stent or bypass 90–99% Critical — very high heart attack risk Urgent treatment 100% Complete blockage (total occlusion) Depends on duration and viable muscle at risk Which Arteries Are Named in the Report LAD (Left Anterior Descending): Supplies the front of the heart. The most important coronary artery — sometimes called the “widow maker” when severely blocked. LCx (Left Circumflex): Supplies the side and back of the left ventricle. RCA (Right Coronary Artery): Supplies the right ventricle and the back of the left ventricle. Left Main: The trunk from which the LAD and LCx both arise. A significant left main blockage is treated as a surgical situation in most cases. What Happens After Coronary Angiography If Blockages Are Found? Single, simple blockage: May be treated with angioplasty and stenting in the same session or shortly after — particularly if the patient is non-diabetic and the anatomy is straightforward. Multiple blockages or complex anatomy: The angiography images are reviewed by a Heart Team — interventional cardiologist and cardiac surgeon together. They use the SYNTAX score to assess complexity and recommend either angioplasty or bypass surgery based on what gives the best long-term result for that specific anatomy. Left main disease: A cardiac surgical review is mandatory before any decision. Bypass surgery is recommended in most left main cases. No significant blockages: Coronary artery disease is effectively ruled out. Your symptoms need evaluation for other causes. You Do Not Have to Decide Immediately One thing many patients do not know: if angiography is being done electively and blockages are found, you are not obligated to accept stenting on the same table. You have every right to review the images, take the report, and seek an independent review of your angiogram from a cardiac surgeon before deciding between bypass surgery and angioplasty. This is not delay — it is good medicine. Frequently Asked Questions — What Is Coronary Angiography What is coronary angiography and is it dangerous? Coronary angiography is a catheter-based diagnostic test that uses dye and X-ray to reveal coronary artery blockages. It is very safe — serious complications occur in less than 0.1% of elective procedures. Local anaesthesia, 30–45 minutes, wrist access in most cases. What does 70% blockage on angiography mean? The artery is narrowed to 30% of its normal diameter — significantly restricting blood flow. Most cardiologists and cardiac surgeons recommend treatment for 70%+ blockages in major coronary arteries. Whether treatment is stenting or bypass surgery depends on the number of vessels involved and the overall anatomy. Is coronary angiography painful? Not painful. The wrist or groin is numbed with local anaesthetic before the catheter is inserted. Patients feel a brief warm flush when the dye is injected — 10–15 seconds — which is normal. Most patients find angiography considerably less uncomfortable than they anticipated. What happens after coronary angiography if a blockage is found? A single simple blockage may be stented in the same session. Multiple

Cardiac Surgery

What Is an Echocardiogram? What It Shows and How to Read Your Report

What Is an Echocardiogram? Echocardiogram is an ultrasound scan of your heart — the single most important investigation in cardiac medicine, and the test Dr. Ved Prakash reviews before making any surgical recommendation. If you have been asked to get one, or if you already have a report in hand and cannot understand it, this guide explains what an echocardiogram shows, how to read the key numbers, and what different findings mean for your treatment. Dr. Ved Prakash is Director of Cardiothoracic and Vascular Surgery at Yatharth Super Speciality Hospitals, Greater Noida, and has been reviewing echocardiograms as part of surgical planning for over 8 years across Medanta, Narayana, and Sarvodaya. What Is an Echocardiogram — In Plain Language An echocardiogram uses sound waves at a frequency too high for human hearing — ultrasound — to produce real-time, moving images of your heart. The probe is placed on your chest and the reflected sound waves are converted into pictures on a screen. You see the heart beating, the walls contracting, the valves opening and closing — all in motion. Unlike an ECG, which only shows electrical activity, an echocardiogram shows structure. It answers the questions an ECG cannot: Is the heart pumping effectively? Are the valves leaking or narrowed? Are any walls of the heart not moving properly? Is there fluid around the heart? A standard echocardiogram takes 20–30 minutes, uses no radiation, and requires no preparation. It is painless. What Does an Echocardiogram Show? A single echocardiogram provides more clinical information about the heart than any other non-invasive test: Ejection fraction (EF) — the pumping power of the left ventricle. The most important number on the report. Wall motion — whether each segment of the heart wall is contracting normally. Areas that are not moving indicate past heart attack damage or ongoing ischaemia. Valve function — whether each of the four valves is opening fully (stenosis) or closing completely (regurgitation), and how severe the problem is. Chamber size — whether the heart chambers have enlarged from chronic pressure or volume overload. Pericardial effusion — fluid around the heart that can compress cardiac function if significant. Congenital defects — holes between chambers (ASD, VSD) or structural abnormalities present from birth. How to Read Your Echocardiogram Report Ejection Fraction (EF) This is the most watched number on any echocardiogram report. It measures what percentage of blood in the left ventricle is pumped out with each heartbeat. 55–70%: Normal. The heart is pumping well. 40–54%: Mildly reduced. Medication review and repeat echocardiogram in 3–6 months is standard. 30–39%: Moderately reduced. Specialist review and treatment required. Below 30%: Severely reduced. Significant heart failure. This number changes what surgery is recommended, when it is done, and how Valve Severity Grading Every abnormal valve on the echocardiogram is graded as mild, moderate, or severe. Mild disease is monitored — repeat echo in 1–2 years. Moderate disease means more frequent follow-up — every 6–12 months. Severe disease with symptoms, or with evidence of chamber enlargement on the same echocardiogram, is a surgical discussion. This is the threshold at which a consultation with a cardiac surgeon — specifically about heart valve surgery — becomes necessary. Wall Motion Abnormality Each segment of the left ventricular wall is scored: 1 = normal movement, 2 = reduced movement (hypokinesia), 3 = no movement (akinesia), 4 = bulging outward (dyskinesia — indicates a ventricular aneurysm). Any score above 1 in multiple segments means past heart attack damage or active ischaemia from a blocked artery. Types of Echocardiogram Transthoracic Echocardiogram (TTE) The standard test. Probe on the chest wall. Completely painless. No preparation. Done in 20–30 minutes. This is what most patients have when their cardiologist orders an “echo”. Transoesophageal Echocardiogram (TOE) A probe is passed into the food pipe under sedation — giving far clearer images of the mitral valve, the back of the heart, and the left atrium. Used before mitral valve surgery, in suspected endocarditis, and to look for clots in the left atrial appendage before cardioversion. Stress Echocardiogram An echocardiogram is taken at rest, then immediately after exercise on a treadmill. Wall motion abnormalities that only appear during exertion reveal blocked arteries that are not obvious on a resting echo — useful when symptoms suggest angina but the resting study is normal. When Does an Echocardiogram Lead to Surgery? Not every abnormal echocardiogram leads to surgery. But the following findings typically trigger a surgical consultation: Ejection fraction below 35% with symptoms of breathlessness or fatigue Severe aortic stenosis — valve area below 1.0 cm² with any symptoms Severe mitral regurgitation with increasing left ventricular dimensions Significant ASD or VSD with right heart enlargement on the same study Multiple wall motion abnormalities suggesting active ischaemia from blocked coronary arteries If your echocardiogram has raised a concern and you want a surgical opinion, you can share your echo report online for a consultation with Dr. Ved Prakash — without having to travel first. Frequently Asked Questions — What Is an Echocardiogram What is an echocardiogram and is it the same as an ECG? No — they are completely different. An ECG records electrical signals. An echocardiogram uses ultrasound to show the heart’s structure and movement — valves, walls, chambers, fluid. An echocardiogram gives far more diagnostic information than an ECG. What does ejection fraction mean on an echocardiogram? Ejection fraction is the percentage of blood pumped out by the left ventricle per beat. Normal is 55–70%. Below 40% indicates significant heart failure and directly influences decisions about surgery type and timing. What does a normal echocardiogram report look like? EF 55–70%, all four valves functioning normally, no wall motion abnormalities, normal chamber sizes, no pericardial effusion. If your report shows all of these, your heart structure and function are normal. Is an echocardiogram safe? Completely safe. Ultrasound only — no radiation. Painless and preparation-free. Safe for newborns, pregnant women, and the elderly without any risk. Dr. Ved Prakash | Director, CTVS — Yatharth Super Speciality Hospitals, Greater Noida 📞

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